By S. Zeerleder, B. Zwart, W. A. Wuillemin, L. A. Aarden, A. B. J. Groeneveld, C. Caliez (auth.), Carl A. Burtis, Mathias M. Müller (eds.)
Advances in severe Care Testing includes scientific and laboratory reviews with regards to significantly unwell sufferers regarding new expertise, remedy, and alertness or interpretation of latest checks. the subject material of the booklet is of curiosity to either clinicians and laboratory scientists with quite a lot of subject matters together with irritation, an infection, pressure, hypoxia, ischaemia, cardiology, haemodynamics, blood gases, electrolytes, hint components, nephrology, gastroenterology, haematology and new applied sciences.
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Additional resources for Advances in Critical Care Testing: The 2002 IFCC-Roche Diagnostics Award
02) (table 2), but both groups did not differ in terms of underlying etiology of septic shock (table 1), sex-ratio, prevalence of acute pancreatitis and chronic renal failure nor in levels of PCT,CRP, WBC and Ca2+ (table 2). Spapen, K. ver Elst, L. Huyghens, F. Gorus Table 1. Origin of septic shock and microbiological findings All (n=53) Survivo rs (n=28) Non su rvivors (n=25) Pneumonia' 37 17 20 Peritonitis" 7 4 3 Urosepsis' 5 4 Type of infection o Catheter sepsis' 2 2 o o Gram-negative 23 12 11 Gram-positive 8 5 3 Mixed (~ 1 type of Gram-negative) 8 5 3 Meningitis' Wound infecrion" Type of path oge n o Anaerobe Viral 2 Yeast 9 3 6 • The diagnosis of pneumonia was based on clinical and chest X-ray findi ngs in all patients.
2. 3. Swope MD, Lolis E. MIF: cytokine, hormone or enzyme? Rev Physiol Biochem Pharmaco l 1999;139:1- 32 Calandra T, Bernhagen J, Metz CN et al. MIF as a glucocorticoid- induced modulator of cytoki ne production . Nature 1995; 377:68-7 1 Beishuizen A. Imm un e-neuro-endocri ne ada pta tion in cri tical ill ness. Th esis. Vrije Universitei t 2001, Amste rdam Introduction Macrophage migration inhibitory factor (MIF) is a multifunctional mediator with pituitary hormone, macrophage-derived cytokine, and catalytic enzyme activities, which appear to be of pivotal importance to the regul ation of the host immune and inflammatory response (1, 2).
GC might exert dual feedback effects at the level of the pituitary gland, inhibiting ACTHsecretion while stimulating MIF secretion. High levelsof MIF may be a crucial factor limiting the immunosuppressive effects of even high dose GC treatment for septic shock or late ARDS (52,53). Anti-MIF therapy in combination with low dose GC may be considered an important treatment option for sepsis. In relative adrenal insufficiency, for which we are still searching for reliable diagnostic tests (54,55), MIF might be the crucial factor in determining which patients will respond to GC therapy.
Advances in Critical Care Testing: The 2002 IFCC-Roche Diagnostics Award by S. Zeerleder, B. Zwart, W. A. Wuillemin, L. A. Aarden, A. B. J. Groeneveld, C. Caliez (auth.), Carl A. Burtis, Mathias M. Müller (eds.)